Patient demographics, clinical information including vascular risk factors, and medical histories were assessed during hospital admission. In order to be eligible for inclusion in the study, the symptom complex of TIA patients had to encompass weakness, speech disturbance, dysarthria or dysphasia for greater than 5 minutes. We enrolled patients aged 40 years or older with documented histories of ischemic stroke or transient ischemic attack (TIA) within 24 hours of symptom onset between January 2018 and December 2018. Therefore, we investigated the relationship between baseline characteristics of acute ischemic stroke and BV within 24 hours of symptom onset in such patients. However, the relationship between BV and biochemical parameters during the acute stage of ischemic stroke have not yet been elucidated. There is evidence to show that BV is significantly higher in cases of lacunar or cardioembolic strokes. Although high BV is known to contribute to stroke occurrence, few studies have examined BV in acute ischemic stroke. Furthermore, BV can be modified using medical treatments such as antithrombotics or statins. BV can also be altered during various physiopathological conditions including obesity, cigarette smoking, chronic heart failure, hypertension, and diabetes. Several clinical and epidemiologic studies have demonstrated an association between BV and the occurrence of major thromboembolic events. High BV, in turn, increases thromboembolic risk and plays an important role in cerebro-cardiovascular diseases. High aggregation and low deformability of RBCs can also increase BV. Increases in these factors correspond to increases in BV. BV is an essential hemorheological factor and is determined by hematocrit, plasma viscosity (PV), and properties of red blood cells (RBCs). Blood viscosity (BV) is defined as the intrinsic resistance of blood to flow and serves as a measure of blood “stickiness”.
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